Journal articlePreclinical

[The Peptide Drug ACTH(4-7)PGP (Semax) Suppresses mRNA Transcripts Encoding Proinflammatory Mediators Induced by Reversible Ischemia of the Rat Brain]

Molekuliarnaia biologiia, 2021 May-Jun. Dergunova LV, Dmitrieva VG, Filippenkov IB and 9 others.

Study type
Journal article
Subjects
Animal / preclinical (no Humans MeSH term)
Journal
Molekuliarnaia biologiia
Published
2021 May-Jun
PubMed
PMID 34097675 ↗
DOI
10.31857/S0026898421010043 ↗

Where PepCue cites this

This paper is part of the evidence base behind this profile. Citing a study is not an endorsement of the compound: papers that found no effect are indexed the same way.

N-Acetyl Semax Amidate →

Indexed under

MeSH terms assigned by the National Library of Medicine.

Adrenocorticotropic HormoneAnimalsBrainBrain IschemiaIschemiaNeuroprotective AgentsPeptide FragmentsPharmaceutical PreparationsRNA, MessengerRatsRats, Wistar

Other papers on N-Acetyl Semax Amidate

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[The efficacy of semax in the tretament of patients at different stages of ischemic stroke]
Journal article · 2011
[The effect of semax and its C-end peptide PGP on expression of the neurotrophins and their receptors in the rat brain during incomplete global ischemia]
Journal article · 2006
Semax, an analogue of adrenocorticotropin (4-10), binds specifically and increases levels of brain-derived neurotrophic factor protein in rat basal forebrain

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Compounds